{"id":17698,"date":"2026-09-21T17:22:48","date_gmt":"2026-09-21T17:22:48","guid":{"rendered":"https:\/\/atlasemhematologia.com.br\/blog\/?p=17698"},"modified":"2026-09-21T17:22:57","modified_gmt":"2026-09-21T17:22:57","slug":"o-hemograma-e-a-policitemia-vera","status":"publish","type":"post","link":"https:\/\/atlasemhematologia.com.br\/blog\/o-hemograma-e-a-policitemia-vera\/","title":{"rendered":"O hemograma e a Policitemia Vera"},"content":{"rendered":"\n<p class=\"wp-block-paragraph\">A Policitemia Vera \u00e9 uma neoplasia mieloproliferativa clonal caracterizada pela produ\u00e7\u00e3o excessiva de hem\u00e1cias, independentemente dos n\u00edveis de eritropoietina. Diferentemente das eritrocitoses secund\u00e1rias, que ocorrem em resposta a hip\u00f3xia ou outros est\u00edmulos fisiol\u00f3gicos, a PV resulta de uma muta\u00e7\u00e3o som\u00e1tica que confere autonomia proliferativa \u00e0s c\u00e9lulas progenitoras hematopo\u00e9ticas. O hemograma \u00e9 o ponto de partida diagn\u00f3stico, e seus achados s\u00e3o frequentemente o primeiro sinal de uma condi\u00e7\u00e3o que, sem tratamento, est\u00e1 associada a risco significativo de trombose, transforma\u00e7\u00e3o para mielofibrose e progress\u00e3o para leucemia mieloide aguda. Reconhecer o padr\u00e3o laboratorial da PV \u00e9 uma das compet\u00eancias mais relevantes da hematologia laboratorial.<\/p>\n\n\n\n<figure class=\"wp-block-image size-full is-resized\"><img fetchpriority=\"high\" decoding=\"async\" width=\"398\" height=\"369\" src=\"https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/09\/image-8.png\" alt=\"\" class=\"wp-image-17699\" style=\"aspect-ratio:1.08;width:432px;height:auto\" srcset=\"https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/09\/image-8.png 398w, https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/09\/image-8-300x278.png 300w\" sizes=\"(max-width: 398px) 100vw, 398px\" \/><\/figure>\n\n\n\n<h3 class=\"wp-block-heading\"><\/h3>\n\n\n\n<h4 class=\"wp-block-heading\"><strong>Achados do hemograma na Policitemia Vera: mais do que eritrocitose<\/strong><\/h4>\n\n\n\n<p class=\"wp-block-paragraph\">O achado central da PV no hemograma \u00e9 a eritrocitose, definida como eleva\u00e7\u00e3o da hemoglobina e\/ou hemat\u00f3crito acima das faixas de refer\u00eancia. Entretanto, a PV \u00e9 uma panmielose, e o hemograma frequentemente reflete o envolvimento das tr\u00eas linhagens. Leucocitose est\u00e1 presente em aproximadamente 49% dos pacientes ao diagn\u00f3stico, e trombocitose em cerca de 53%. Portanto, a associa\u00e7\u00e3o de eritrocitose com leucocitose e trombocitose deve sempre levantar suspeita de neoplasia mieloproliferativa, em especial de PV.<\/p>\n\n\n\n<p class=\"wp-block-paragraph\">A morfologia eritrocit\u00e1ria no esfrega\u00e7o \u00e9 geralmente normal nas fases iniciais. Entretanto, quando h\u00e1 defici\u00eancia de ferro concomitante, situa\u00e7\u00e3o comum em pacientes com PV que realizam flebotomias terap\u00eauticas repetidas, podem surgir microcitose e hipocromia, que &#8220;mascaram&#8221; a eritrocitose e dificultam a interpreta\u00e7\u00e3o do hemograma. A basofilia leve e a eosinofilia discreta tamb\u00e9m podem estar presentes, compondo o padr\u00e3o de panmielose caracter\u00edstico da doen\u00e7a.<\/p>\n\n\n\n<h3 class=\"wp-block-heading\"><\/h3>\n\n\n\n<h4 class=\"wp-block-heading\"><strong>Eritrocitose prim\u00e1ria versus secund\u00e1ria: como o laborat\u00f3rio auxilia na diferencia\u00e7\u00e3o<\/strong><\/h4>\n\n\n\n<p class=\"wp-block-paragraph\">A identifica\u00e7\u00e3o de eritrocitose no hemograma exige diferencia\u00e7\u00e3o entre causa prim\u00e1ria, como PV, e causas secund\u00e1rias, como hip\u00f3xia cr\u00f4nica por apneia do sono, tabagismo, doen\u00e7a pulmonar obstrutiva cr\u00f4nica, uso de andr\u00f3genos e neoplasias produtoras de eritropoietina. Essa diferencia\u00e7\u00e3o tem implica\u00e7\u00e3o direta no manejo cl\u00ednico e n\u00e3o pode ser feita apenas pelo hemograma.<\/p>\n\n\n\n<p class=\"wp-block-paragraph\">A dosagem s\u00e9rica de eritropoietina \u00e9 o exame de triagem mais importante nesse contexto: valores subnormais sugerem produ\u00e7\u00e3o aut\u00f4noma de hem\u00e1cias, como ocorre na PV, enquanto valores normais ou elevados apontam para causas secund\u00e1rias. A pesquisa da muta\u00e7\u00e3o JAK2 V617F \u00e9 o exame confirmat\u00f3rio de maior peso: presente em mais de 95% dos pacientes com PV, sua detec\u00e7\u00e3o praticamente confirma o diagn\u00f3stico quando associada \u00e0 eritrocitose expressiva. Nos casos com JAK2 V617F negativo, a pesquisa das muta\u00e7\u00f5es no \u00e9xon 12 do gene JAK2 est\u00e1 indicada.<\/p>\n\n\n\n<figure class=\"wp-block-image size-full is-resized\"><img decoding=\"async\" width=\"401\" height=\"382\" src=\"https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/09\/image-9.png\" alt=\"\" class=\"wp-image-17700\" style=\"aspect-ratio:1.0503597122302157;width:438px;height:auto\" srcset=\"https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/09\/image-9.png 401w, https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/09\/image-9-300x286.png 300w\" sizes=\"(max-width: 401px) 100vw, 401px\" \/><\/figure>\n\n\n\n<h3 class=\"wp-block-heading\"><\/h3>\n\n\n\n<h4 class=\"wp-block-heading\"><strong>Complica\u00e7\u00f5es e monitoramento laboratorial<\/strong><\/h4>\n\n\n\n<p class=\"wp-block-paragraph\">A trombose \u00e9 a principal causa de morbidade e mortalidade na PV, afetando vasos arteriais e venosos. O laborat\u00f3rio tem papel no monitoramento dos fatores de risco: o hemat\u00f3crito acima de 45% est\u00e1 associado a maior risco tromb\u00f3tico, e seu controle abaixo desse limiar \u00e9 um objetivo terap\u00eautico estabelecido. A evolu\u00e7\u00e3o para mielofibrose p\u00f3s-PV, que ocorre em cerca de 9% dos pacientes ao longo de sete anos, \u00e9 sinalizada laboratorialmente pelo surgimento progressivo de anemia, dacri\u00f3citos no esfrega\u00e7o e padr\u00e3o leucoeritrobl\u00e1stico. A transforma\u00e7\u00e3o para leucemia mieloide aguda, embora menos frequente, deve ser suspeitada diante de piora abrupta das citopenias ou surgimento de blastos no sangue perif\u00e9rico.<\/p>\n\n\n\n<p class=\"wp-block-paragraph\"><\/p>\n\n\n\n<figure class=\"wp-block-image size-large\"><a href=\"https:\/\/eventos.atlasemhematologia.com.br\/listadeesperatimeblogatlas\"><img decoding=\"async\" width=\"1030\" height=\"276\" src=\"https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/03\/BANNER-BLOG-ATLAS-1030x276.png\" alt=\"\" class=\"wp-image-17314\" srcset=\"https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/03\/BANNER-BLOG-ATLAS-1030x276.png 1030w, https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/03\/BANNER-BLOG-ATLAS-300x80.png 300w, https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/03\/BANNER-BLOG-ATLAS-768x206.png 768w, https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/03\/BANNER-BLOG-ATLAS-1536x411.png 1536w, https:\/\/atlasemhematologia.com.br\/blog\/wp-content\/uploads\/2026\/03\/BANNER-BLOG-ATLAS-2048x549.png 2048w\" sizes=\"(max-width: 1030px) 100vw, 1030px\" \/><\/a><\/figure>\n\n\n\n<h3 class=\"wp-block-heading\"><\/h3>\n\n\n\n<p class=\"wp-block-paragraph\"><strong>Refer\u00eancias<\/strong><\/p>\n\n\n\n<p class=\"wp-block-paragraph\">OLIVEIRA, Larissa Santana; ALCANTARA, Thiago Ruan de Lima. <em>Atlas em hematologia: um guia visual para a identifica\u00e7\u00e3o de c\u00e9lulas sangu\u00edneas<\/em>. 1. ed. Salvador: Oxente, 2025.<\/p>\n\n\n\n<p class=\"wp-block-paragraph\">MORETTI, Marcelo Pasquali et al. Policitemia vera: relato de caso. <strong>Arquivos catarinenses de medicina<\/strong>, v. 37, n. 3, p. 76-9, 2008.&nbsp;<\/p>\n\n\n\n<p class=\"wp-block-paragraph\">REEVES, Brandi N. et al. Ropeginterferon alfa-2b-njft treatment in essential thrombocythemia across different driver mutations: results from a North American, single-arm, multicentre study (EXCEED-ET). <strong>The Lancet Regional Health\u2013Americas<\/strong>, v. 61, 2026.&nbsp;<\/p>\n\n\n\n<p class=\"wp-block-paragraph\">TREMBLAY, Douglas et al. Diagnosis and treatment of polycythemia vera: a review. <strong>Jama<\/strong>, v. 333, n. 2, p. 153-160, 2025.<\/p>\n","protected":false},"excerpt":{"rendered":"<p>A Policitemia Vera \u00e9 uma neoplasia mieloproliferativa clonal caracterizada pela produ\u00e7\u00e3o excessiva de hem\u00e1cias, independentemente dos n\u00edveis de eritropoietina. Diferentemente das eritrocitoses secund\u00e1rias, que ocorrem em resposta a hip\u00f3xia ou outros est\u00edmulos fisiol\u00f3gicos, a PV resulta de uma muta\u00e7\u00e3o som\u00e1tica que confere autonomia proliferativa \u00e0s c\u00e9lulas progenitoras hematopo\u00e9ticas. O hemograma \u00e9 o ponto de partida [&hellip;]<\/p>\n","protected":false},"author":6,"featured_media":17701,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"pagelayer_contact_templates":[],"_pagelayer_content":"","footnotes":""},"categories":[4],"tags":[],"class_list":["post-17698","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-hemograma"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.2 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>O hemograma e a Policitemia Vera - Blog - Atlas em Hematologia<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/atlasemhematologia.com.br\/blog\/o-hemograma-e-a-policitemia-vera\/\" \/>\n<meta property=\"og:locale\" content=\"pt_BR\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"O hemograma e a Policitemia Vera - Blog - Atlas em Hematologia\" \/>\n<meta property=\"og:description\" content=\"A Policitemia Vera \u00e9 uma neoplasia mieloproliferativa clonal caracterizada pela produ\u00e7\u00e3o excessiva de hem\u00e1cias, independentemente dos n\u00edveis de eritropoietina. Diferentemente das eritrocitoses secund\u00e1rias, que ocorrem em resposta a hip\u00f3xia ou outros est\u00edmulos fisiol\u00f3gicos, a PV resulta de uma muta\u00e7\u00e3o som\u00e1tica que confere autonomia proliferativa \u00e0s c\u00e9lulas progenitoras hematopo\u00e9ticas. 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